منابع مشابه
Conspecific injury raises an alarm in medaka
In the late 1930s, Karl von Frisch reported that semiochemicals released upon injury, act as alarm substances (Schreckstoff) in fish. In Ostariophysi species, club cells in the epidermis are believed to contain cues related to alarm substance; however, the function of club cells, primarily as reservoirs of alarm substance has been debated. Here, I describe an alarm response in the Japanese rice...
متن کاملCytokines (IL-17, IL-23, and IL-33) in Systemic lupus erythematosus in Trinidad and Tobago.
Systemic lupus erythematosus (SLE) is the most common autoimmune disease. It is characterized by the presence of hundreds of autoantibodies against many organs and tissues, including the presence of a large number of autoantibodies, which are specific to self-antigens mainly of nuclear origin such as Smith antigen, double-stranded DNA (dsDNA), anti-Sjögren’s syndrome-related antigen A and B (SS...
متن کاملIL-33 and Airway Inflammation
Interleukin-33 (IL-33) is the 11th member of IL-1 cytokine family which includes IL-1 and IL-18. Unlike IL-1β and IL-18, IL-33 is suggested to function as an alarmin that is released upon endothelial or epithelial cell damage and may not enhance acquired immune responses through activation of inflammasome. ST2, a IL-33 receptor component, is preferentially expressed by T-helper type (Th) 2 cell...
متن کاملتغییرات اینترلوکین 33 (il-33) و اینترلوکین 23 (il-23) در بیماران مبتلا به آرتریت روماتوئید
پیش زمینه: آرتریت روماتوئید یک بیماری خودایمنی سیستمیک مزمن است که عمدتا به وسیله پاسخ های التهابی که روی مفاصل و بافت های اطراف آنها اثر می گذارند شناخته می شود. با وجودی که دلیل ابتلا به آرتریت روماتوئید ناشناخته است چنین به نظر می رسد که سیتوکین هایی که به وسیله سلول های مختلف مثل لنفوسیت ها، مونوسیت ها، سلول های اپی تلیال و اندوتلیال تولید می شوند نقش های مهمی را در القا و پیشبرد پاسخ های ا...
15 صفحه اولMeasurement of Interleukin-33 (IL-33) and IL-33 Receptors (sST2 and ST2L) in Patients with Rheumatoid Arthritis
The interleukin-33 (IL-33)/ST2 pathway has emerged as an intercellular signaling system that participates in antigen-allergen response, autoimmunity and fibrosis. It has been suggested that IL-33/ST2 signaling has been involved in the pathogenesis of rheumatoid arthritis (RA), because IL-33 and its receptor have been specifically mapped to RA synovium. The aim of this study was to determine the...
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ژورنال
عنوان ژورنال: Immunity
سال: 2009
ISSN: 1074-7613
DOI: 10.1016/j.immuni.2009.06.011